Now reading Entry 049 / 102 Last revised Jan 22, 2026 15 sources 3 comparisons Methodology →
A specimen of the Metabolic drawer Drawer A · Metabolic

Mazdutide

Investigational

A dual GLP-1 and glucagon receptor agonist developed by Innovent/Eli Lilly. NMPA approved in China (2025) for obesity. The GLORY-1 trial showed 14.0% weight loss at 48 weeks with the 6mg dose. The DREAMS-3 head-to-head trial vs semaglutide for combined diabetes + obesity outcomes is ongoing, with results not yet published.

IBI362 · LY3305677 · Xinermei

Research evidence
High

14 human studies

Preclinical
7%
Clinical
93%

Based on 15 cited sources

Evidence Score71/100
Emerging / moderate
Research Depth90/100
Mechanism80/100
Plausibility88/100
Global Coverage54/100
Community Experience16/100
Effectiveness85/100

clinically demonstrated · high confidence

Demonstrated effect magnitude — not a recommendation or safety claim.

Research Depth 90: highest tier is meta-analysis of multiple independent double-blind placebo-controlled Phase 3 RCTs (1A 35) — GLORY-1 N=610 (40421736), plus a Phase 2 program (37943529 N=249; 38092790 N=248); best studies are low-RoB large RCTs with supporting reviews (1B 22), total human N well over 1,000 (1C 14), direct obese/T2D population + SC route + weight and HbA1c outcomes (1D 14), and a modest preclinical base (1E 5). Mechanism 80: named GLP-1R + GCGR targets with binding and functional agonist confirmation (2A 30), receptor-pathway-to-effect largely mapped (2B 24), multi-dose human + in-vivo dose-response (2C 16), confirmed in vivo (40479843 db/db mice) and in humans (2D 10). Plausibility 88: full mechanism->surrogate (weight/HbA1c)->clinical-outcome chain proven in RCTs (3A 34), fully coherent with incretin/glucagon biology (3B 24), multiple same-class agonists reproduce the effect (3C 18), tightly scoped metabolic claims (3D 12). Global Coverage 54: replicated across many Chinese hospital groups but primary trials are almost entirely China/Innovent-led, limiting independent cross-country replication (4A 16, 4B 10); 20+ diverse studies (4C 18); single-regulator NMPA approval (4D 10). Community Experience 16: dossier records negligible documented real-world use; newly approved 2025 so short track record and no consistent user-report corpus (5A 4, 5B 6, 5C 0, 5D 6 for documented trial-level GI tolerability, not a safety claim). Effectiveness basis clinical (high): GLORY-1 6mg -14.0% body weight vs ~0% placebo at 48 weeks, far exceeding the ~5% obesity MCID with hard patient-relevant weight and cardiometabolic outcomes; superior to dulaglutide on HbA1c/weight, though head-to-head vs semaglutide (DREAMS-3, 41260459) is pending and indirect comparisons place it below retatrutide.

Scored June 2026 How we rate →
Evidence Level
high
Not approved for human use by any regulatory agency
Limited human clinical trial data
Consult a healthcare provider before use
Not FDA Approved WADA Prohibited
Identity
SCALE · 1:1 N-TERMINUS C-TERMINUS 30 AA · ~4,000 Da
Also Known As
IBI362 • LY3305677 • Xinermei
Class
Modified peptide
Length
30 amino acids
Mol. weight
~4,000 Da
Sequence
Modified GLP-1 analog with fatty acid side chain
Molecular Structure
H
A
E
G
T
F
T
S
D
V
Hydrophobic
Polar
Positive
Negative

Mazdutide is a dual GLP-1 and glucagon receptor agonist with robust Phase 3 clinical trial data. Its mechanisms are well-characterized in human studies.

How It Works (Simplified)

GLP-1 receptor activation in the brain increases satiety signals, reducing hunger and food intake similar to semaglutide.

Liver Fat Burning

Glucagon receptor activation directly stimulates hepatic fatty acid oxidation, burning fat stored in the liver.

Insulin Enhancement

Glucose-dependent insulin secretion improves blood sugar control without causing hypoglycemia when blood sugar is normal.

Energy Expenditure

Glucagon increases metabolic rate and calorie burning, contributing to weight loss beyond appetite reduction alone.

Key Research: Ji L et al. (NEJM 2025) demonstrated 14.0% weight loss at 48 weeks with the 6mg dose, alongside beneficial effects on all prespecified cardiometabolic measures, in the GLORY-1 trial. PMID:40421736

Important Limitations

  • Currently only approved in China (NMPA); not yet FDA approved
  • Long-term safety data beyond 48 weeks is limited
  • Head-to-head data vs tirzepatide not yet available
  • Optimal patient selection criteria still being refined
  • GI tolerability requires careful dose titration
i. GLP-1 Receptor Pathway · Appetite & Glucose
MazdutideGLP-1R activationBrain satiety centersReduced food intakePancreatic beta cellsInsulin secretionGastric emptyingDelayed nutrient absorption
ii. Glucagon Receptor Pathway · Liver & Metabolism
MazdutideGCGR activationHepatic fatty acid oxidationLiver fat reductionGluconeogenesis modulationBalanced by insulinThermogenesisIncreased energy expenditure
Mechanism GLP-1 receptor activation reducing appetite and improving glycemic control
Established 12 direct studies
Benefit shown to promote significant weight loss
Evidence Level
High
10 Human
2 Animal
1 In Vitro
Mechanism Glucagon receptor activation increasing hepatic fatty acid oxidation
Established 8 direct studies
Benefit shown to reduce liver fat content
Evidence Level
High
6 Human
2 Animal
2 In Vitro
Mechanism Dual receptor agonism improving insulin secretion and gluconeogenesis balance
Established 10 direct studies
Benefit shown to improve glycemic control in type 2 diabetes
Evidence Level
High
8 Human
2 Animal
Mechanism Glucagon-mediated increase in energy expenditure
Supported 2 direct studies
Benefit appears to increase metabolic rate
Evidence Level
Moderate
2 Animal
Mechanism Confidence
Established
Supported
Emerging
Evidence Level
High
Moderate
Low
Very Low
Phase 01 1
Week 1-4

Initial dose titration period. Nausea and GI effects most common during this phase. Early appetite suppression and reduced food intake typically begin within days of first dose.

PMID:40421736
Phase 02 2
Week 4-12

Continued dose escalation to target dose. Steady weight loss averaging 1-2% body weight per month. Blood glucose improvements measurable. GI side effects typically diminish.

PMID:40421736
Phase 03 3
Week 12-24

Maintenance dosing established. Weight loss continues at consistent rate. HbA1c improvements reach clinically significant levels and cardiometabolic measures continue to improve.

PMID:40421736
Phase 04 4
Week 24-48

Peak efficacy period. GLORY-1 showed 14.0% total body weight loss at 48 weeks with the 6mg dose. Sustained glycemic control and improvements across cardiometabolic measures.

PMID:40421736

Research-based observations

This timeline reflects observations from published clinical and preclinical studies. Individual responses may vary significantly. This is not a guarantee of effects or a dosing schedule. Consult qualified healthcare providers for personalized guidance.

Good Signs (6 indicators)
Prescription-only pharmaceutical product from licensed manufacturer
Proper cold chain storage maintained
Clear, colorless solution in pre-filled pen or vial
Intact tamper-evident seal
Within expiration date with proper lot number
Accompanied by official prescribing information
Warning Signs (5 indicators)
Obtained outside regulatory channels
Missing batch/lot documentation
Unclear storage history
Pricing significantly below market rate
No prescribing information included
Bad Signs (6 indicators)
Cloudy, discolored, or particulate-containing solution
Damaged or previously opened packaging
No cold chain verification
Missing manufacturer identification
Expired product
Sourced from unverified or underground suppliers
Positive quality indicator
Requires evaluation
Potential quality issue

For Research Evaluation Only

These quality indicators are general guidelines based on typical peptide characteristics. Professional laboratory testing (HPLC, mass spectrometry) provides definitive quality verification. This checklist is for initial visual evaluation only.

Synergistic
Compatible
Caution
Avoid

Both affect GI function. Mazdutide slows gastric emptying while BPC-157 is gastroprotective. Monitor GI symptoms if combined; interaction data unavailable.

Both are GLP-1 receptor agonists. Combining would duplicate mechanisms and increase risk of severe GI adverse effects and hypoglycemia. No clinical rationale for combination.

Both target GLP-1 receptors with overlapping mechanisms. Combining incretin therapies is contraindicated due to additive effects and safety concerns.

Both are dual GLP-1/glucagon receptor agonists with identical mechanisms. Combination would be redundant and potentially dangerous.

Research Note: Interaction data is based on published literature, mechanistic understanding, and theoretical considerations. Most peptide combinations lack direct clinical study. This information is for educational purposes only and does not constitute medical advice. Always consult qualified healthcare providers.

This dossier synthesizes available evidence from peer-reviewed literature, regulatory documents, and clinical trial registries. Evidence strength ratings follow a modified GRADE approach.

For complete methodology details, see our Methodology page.

Important Disclaimer

This dossier is for educational purposes only and does not constitute medical advice. Always consult a qualified healthcare provider before making health decisions.

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