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CagriSema

Investigational

A fixed-dose combination of cagrilintide (amylin analog) and semaglutide (GLP-1 agonist) for chronic weight management. Phase 3 REDEFINE trials demonstrate 20-23% weight loss at 68 weeks. NDA submitted to FDA December 2025. First GLP-1/amylin combination therapy.

Cagrilintide-Semaglutide · NNC0174-0833

Research evidence
High

16 human studies

Preclinical
11%
Clinical
89%

Based on 18 cited sources

Evidence Score76/100
Emerging / moderate
Research Depth83/100
Mechanism91/100
Plausibility92/100
Global Coverage67/100
Community Experience30/100
Effectiveness82/100

clinically demonstrated · high confidence

Demonstrated effect magnitude — not a recommendation or safety claim.

Research depth: two large adequately-powered phase 3 RCTs (REDEFINE-1 n=3,417; REDEFINE-2 n=1,206) plus phase 2 dose-finding, no published meta-analysis yet. Mechanism: GLP-1R and amylin/calcitonin receptor targets validated by cryo-EM binding plus functional agonism. Plausibility: full mechanism-to-weight-loss chain supported and coherent with incretin-class biology. Global coverage: 10-12 countries but predominantly one sponsor; NDA under review, not yet approved. Community experience: negligible niche discussion for an investigational agent. Effectiveness: clinical basis, ~20% weight loss (large effect, high confidence), tempered by REDEFINE-4 non-inferiority miss vs tirzepatide.

Scored May 2026 How we rate →
Evidence Level
high
Not approved for human use by any regulatory agency
Limited human clinical trial data
Consult a healthcare provider before use
Not FDA Approved WADA Prohibited
Identity
SCALE · 1:1 N-TERMINUS C-TERMINUS 70 AA · ~8,335 Da (combined)
Also Known As
Cagrilintide-Semaglutide • NNC0174-0833
Class
Fixed-dose combination (two peptides)
Length
70 amino acids
Mol. weight
~8,335 Da (combined)
Sequence
Cagrilintide (39 aa) + Semaglutide (31 aa)
Molecular Structure
K
C
N
T
A
T
C
A
T
Q
R
L
A
E
L
S
S
N
L
V
G
S
N
T
P
V
S
S
P
P
L
A
A
T
K
L
L
Q
Y
H
Aib
E
G
T
F
T
S
D
V
S
S
Y
L
E
G
Q
A
A
K
E
F
I
A
W
L
V
R
G
R
G
Hydrophobic
Polar
Positive
Negative

CagriSema combines two distinct appetite-suppressing peptide hormones into a single weekly injection, targeting complementary pathways in the brain.

How It Works (Simplified)

Dual Satiety Signals

GLP-1 (semaglutide) and amylin (cagrilintide) activate separate “stop eating” pathways in the brain, creating a stronger combined appetite suppression effect.

Distinct Brain Regions

Semaglutide targets the hypothalamus (hunger center) while cagrilintide additionally affects the hindbrain and septum (satiety and reward areas).

Synergistic Effect

Clinical trials show the combination produces MORE weight loss than adding the two drugs together would predict, indicating true pharmacological synergy.

For people with prediabetes or diabetes, the combination improves blood sugar control more effectively than either component alone.

Key Research: Garvey WT et al. demonstrated in REDEFINE-1 that CagriSema produced 20.4% mean weight loss at 68 weeks versus 3.0% with placebo in adults with overweight or obesity. PMID:40544433

NDA Submission & REDEFINE Program (2025-2026)

NDA Filing: Novo Nordisk filed a New Drug Application for CagriSema in December 2025, based on the REDEFINE clinical program combining cagrilintide (amylin analog) with semaglutide.

REDEFINE 4 — Head-to-Head vs Tirzepatide: The REDEFINE 4 trial did not meet its primary endpoint of non-inferiority to tirzepatide, with CagriSema achieving 23% weight loss compared to 25.5% with tirzepatide at the highest dose. This result raised questions about CagriSema’s competitive positioning against established dual-agonist therapies.

Next Steps: Novo Nordisk has announced plans for a higher-dose CagriSema trial in H2 2026, aiming to improve upon the REDEFINE 4 results and strengthen the regulatory package.

Important Limitations

  • FDA approval pending (NDA submitted December 2025)
  • REDEFINE 4 failed non-inferiority vs tirzepatide (23% vs 25.5% weight loss)
  • Long-term safety data beyond 68 weeks not yet available
  • Cardiovascular outcomes trial (REDEFINE-3) still ongoing
  • GI side effects occur in approximately 80% of patients (mostly mild-moderate, transient)
  • Weight maintenance after discontinuation unknown
i. GLP-1 Pathway (Semaglutide)
SemaglutideGLP-1R activationHypothalamus
POMC/CART activationsatiety increase
NPY/AgRP inhibitionhunger decrease
Glucose-dependent insulin secretion
ii. Amylin Pathway (Cagrilintide)
CagrilintideAMY1R/AMY2R/AMY3R/CTR activationHindbrain
Area postrema signalingmeal termination
Nucleus tractus solitariussatiety
Reduced postprandial glucagon
Mechanism Dual activation of GLP-1 and amylin receptor pathways in hypothalamus and hindbrain
Established 12 direct studies
Benefit shown to produce significant weight loss exceeding single-agent therapy
Evidence Level
High
4 Human
2 Animal
1 In Vitro
Mechanism GLP-1 receptor activation enhances glucose-dependent insulin secretion and suppresses glucagon
Established 8 direct studies
Benefit shown to improve glycemic control in type 2 diabetes
Evidence Level
High
3 Human
1 Animal
2 In Vitro
Mechanism Amylin receptor activation in area postrema and nucleus tractus solitarius promotes satiety signaling
Established 6 direct studies
Benefit shown to reduce appetite and food intake through complementary brain pathways
Evidence Level
High
4 Human
3 Animal
1 In Vitro
Mechanism Delayed gastric emptying through both GLP-1 and amylin pathways (additive effect)
Supported 4 direct studies
Benefit appears to prolong post-meal satiety and reduce caloric intake
Evidence Level
Moderate
2 Human
2 Animal
Mechanism Confidence
Established
Supported
Emerging
Evidence Level
High
Moderate
Low
Very Low
Phase 01 1
Week 1-4

Dose titration phase. GI side effects (nausea, vomiting, diarrhea) most common during initiation. Appetite suppression typically begins within first 1-2 weeks.

PMID:40544433
Phase 02 2
Week 4-16

Continued dose escalation to maintenance dose. Weight loss acceleration observed. GI side effects typically diminish as tolerance develops.

PMID:40544433
Phase 03 3
Week 16-32

Maintenance phase with ongoing weight loss as participants progress toward the trial's 68-week endpoint.

PMID:40544433
Phase 04 4
Week 32-68

Continued weight loss with plateau approaching. Mean weight loss of 20.4% achieved at 68 weeks in REDEFINE-1. Cardiometabolic improvements sustained.

PMID:40544433

Research-based observations

This timeline reflects observations from published clinical and preclinical studies. Individual responses may vary significantly. This is not a guarantee of effects or a dosing schedule. Consult qualified healthcare providers for personalized guidance.

Good Signs (6 indicators)
FDA-approved pharmaceutical product (pending approval)
Single-use prefilled injection pen from licensed pharmacy
Clear manufacturer labeling with lot number and expiration date
Proper cold chain storage documentation
Prescription from licensed healthcare provider
Clear, colorless solution without particulates
Warning Signs (5 indicators)
Product obtained outside of regulated pharmacy channels
Missing or unclear labeling information
Storage temperature not maintained during shipping
Approaching expiration date
Unusual packaging or labeling inconsistencies
Bad Signs (6 indicators)
Product from unverified or overseas sources
Cloudy solution or visible particles
Damaged or compromised packaging
No prescription or obtained without medical oversight
Significant price discrepancy from market rates
Missing cold chain verification
Positive quality indicator
Requires evaluation
Potential quality issue

For Research Evaluation Only

These quality indicators are general guidelines based on typical peptide characteristics. Professional laboratory testing (HPLC, mass spectrometry) provides definitive quality verification. This checklist is for initial visual evaluation only.

Synergistic
Compatible
Caution
Avoid

Both affect GI function. BPC-157 is gastroprotective while semaglutide component slows gastric emptying. Monitor GI symptoms if combined.

CagriSema already contains semaglutide. Combining with additional semaglutide would result in overdose. Do not use concurrently.

Both are GLP-1 receptor agonists for weight management. Concurrent use would cause overlapping mechanisms and increased adverse effects.

Liraglutide is a GLP-1 agonist. Combining with CagriSema would duplicate GLP-1 pathway activation and increase GI adverse effects.

Research Note: Interaction data is based on published literature, mechanistic understanding, and theoretical considerations. Most peptide combinations lack direct clinical study. This information is for educational purposes only and does not constitute medical advice. Always consult qualified healthcare providers.

18 Sources 16 Human 2 Preclinical

Full reference list available on request. All citations link to PubMed for verification.

This dossier synthesizes available evidence from peer-reviewed literature, regulatory documents, and clinical trial registries. Evidence strength ratings follow a modified GRADE approach.

For complete methodology details, see our Methodology page.

Important Disclaimer

This dossier is for educational purposes only and does not constitute medical advice. Always consult a qualified healthcare provider before making health decisions.

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