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A specimen of the Cognitive drawer Drawer E · Cognitive

Selank

Research Only

A synthetic heptapeptide derived from the immunomodulatory peptide tuftsin, developed by Russian researchers and approved in Russia since 2009 for anxiety and neurasthenia. Demonstrates anxiolytic effects comparable to benzodiazepines without sedation, cognitive impairment, or dependence liability. Extensive Russian clinical research supports efficacy, though Western independent replication remains limited.

TP-7 · Selanc · Threonyl-Lysyl-Prolyl-Arginyl-Prolyl-Glycyl-Proline

Research evidence
Moderate

5 human studies

Preclinical
80%
Clinical
20%

Based on 25 cited sources

Evidence Score67/100
Emerging / moderate
Research Depth63/100
Mechanism70/100
Plausibility82/100
Global Coverage44/100
Community Experience71/100
Effectiveness55/100

clinically demonstrated · low confidence

Demonstrated effect magnitude — not a recommendation or safety claim.

Research Depth 63: best human evidence is a single adequately-powered active- comparator RCT (Zozulia 2008, PMID:18454096, n=62 Selank vs medazepam, 1A=28) plus a placebo-controlled human fMRI study (PMID:32342318, n=52); Russian small-RCT methods rated "some concerns" (1B=10), total human N in the 50-300 band (1C=6), direct anxiety population/route/outcome (1D=12), and a substantial single-cluster preclinical program (1E=7). Mechanism 70: measured enkephalinase inhibition (IC50 ~15 uM, PMID:11550013) plus functional GABA-A subunit gene-expression modulation (PMID:26924987) gives binding+functional confirmation without knockout (2A=30); most of the proximal chain mapped (2B=18); dose-response in vitro and in vivo (2C=13); confirmed in mammalian in-vivo models (2D=9). Plausibility 82: mechanism->surrogate->clinical anxiety benefit largely supported (3A=30), fully coherent with GABAergic/enkephalinergic anxiety biology (3B=24), strong same-class analogy to benzodiazepines (3C=17), moderately broad claim set spanning anxiolysis/cognition/mood/immune (3D=11). Global Coverage 44: replication confined to a Moscow institutional cluster (4A=12), 1-2 countries (4B=8), ~25 diverse studies (4C=14), Russian regulatory approval since 2009 (4D=10). Community Experience 71: broad sustained nootropic- community use >7 years, broadly consistent "anxiolysis without sedation" themes, no recurring serious adverse signal (5A=22/5B=24/5C=14/5D=11). Effectiveness clinical basis: one small Russian RCT shows anxiolytic effect comparable to medazepam with HAM-A reductions (E1~18/E2~14/E3~13/E4~12 ≈ 55), confidence low given single small non-Western RCT and no independent Western replication.

Scored June 2026 How we rate →
~
Evidence Level
moderate
Not approved for human use by any regulatory agency
Limited human clinical trial data
Consult a healthcare provider before use
Not FDA Approved WADA Prohibited
Identity
SCALE · 1:1 N-TERMINUS C-TERMINUS 7 AA · 751.9 Da
Also Known As
TP-7 • Selanc • Threonyl-Lysyl-Prolyl-Arginyl-Prolyl-Glycyl-Proline
Class
Heptapeptide (Tuftsin Analogue)
Length
7 amino acids
Mol. weight
751.9 Da
Sequence
TKPRPGP
Molecular Structure
T
K
P
R
P
G
P
Hydrophobic
Polar
Positive
Negative

Selank is a synthetic heptapeptide derived from the endogenous immunomodulatory peptide tuftsin (Thr-Lys-Pro-Arg) with a C-terminal Pro-Gly-Pro extension that confers CNS activity and enhanced stability. Unlike traditional anxiolytics that directly bind receptors, Selank works primarily through gene expression modulation, producing sustained effects without tolerance or dependence.

How It Works (Simplified)

GABA Enhancement

Modulates gene expression of GABA-A receptor subunits, enhancing inhibitory neurotransmission for calming effects without the sedation or dependence of benzodiazepines.

Enkephalin Protection

Inhibits enzymes that degrade enkephalins, allowing your natural “feel-good” opioids to work longer for mood elevation and stress resilience.

BDNF Boost

Increases brain-derived neurotrophic factor in the hippocampus and frontal cortex, supporting memory consolidation, learning, and neuroplasticity.

Serotonin Balance

Modulates 5-HT1A and 5-HT2A receptor expression, contributing to mood stabilization and anxiolytic effects similar to buspirone but at the transcriptional level.

Key Research: Zozulia AA et al. (2008) demonstrated anxiolytic efficacy comparable to medazepam in 62 patients with generalized anxiety disorder and neurasthenia, with additional antiasthenic effects. PMID:18454096

Important Limitations

  • Most clinical research originates from Russian institutions with limited Western replication
  • Approved in Russia (2009) but not by FDA, EMA, or other Western regulatory agencies
  • Many primary studies published only in Russian, limiting accessibility
  • Long-term effects beyond 14-day treatment courses less well characterized
  • Gene expression mechanism means onset takes days rather than minutes
i. GABAergic System Modulation · Primary Anxiolytic Mechanism
SelankGene expression modulationGABA-A receptor subunit changes (α, β, γ)
Enhanced GABAergic neurotransmission
Reduced neuronal excitability
Anxiolytic effect without benzodiazepine-like sedation/tolerance
ii. Enkephalinergic System · Mood & Well-being
SelankEnkephalin-degrading enzyme inhibitionElevated endogenous enkephalin
Mu and delta opioid receptor activation
Natural mood enhancement
Stress resilience without exogenous opioid risks
iii. Neurotrophic Pathway · Cognitive Enhancement
SelankFrontal cortex & hippocampusBDNF mRNA upregulation
Neuroplasticity enhancement
Memory consolidation improvement
Stress-induced cognitive decline protection
Mechanism GABA-A receptor subunit gene expression modulation
Established 8 direct studies
Benefit shown to reduce anxiety without sedation or dependence
Evidence Level
Moderate
4 Human
12 Animal
3 In Vitro
Mechanism Enkephalin-degrading enzyme inhibition elevating endogenous opioids
Supported 5 direct studies
Benefit appears to enhance natural mood regulation and stress resilience
Evidence Level
Moderate
2 Human
6 Animal
4 In Vitro
Mechanism BDNF upregulation in hippocampus and frontal cortex
Supported 4 direct studies
Benefit may improve memory consolidation and cognitive function
Evidence Level
Low
1 Human
8 Animal
2 In Vitro
Mechanism Serotonergic and monoaminergic system modulation
Supported 4 direct studies
Benefit may stabilize mood and reduce depressive symptoms
Evidence Level
Low
1 Human
5 Animal
2 In Vitro
Mechanism Confidence
Established
Supported
Emerging
Evidence Level
High
Moderate
Low
Very Low

Reported positives

  • Reduced anxiety without sedation frequently reported
  • Improved focus and cognitive clarity noted
  • Better stress management and emotional stability
  • Nasal administration convenient

Reported negatives

  • Effects subtle compared to pharmaceutical anxiolytics
  • Short duration of action
  • Some users report fatigue at higher amounts
  • Limited supplier quality control

“Often compared to and stacked with semax. Favored for anxiolytic effects in nootropic communities.”

Self-reported, unverified accounts — not clinical evidence and no substitute for the cited research. Anecdotes are prone to selection bias and placebo effects.

Phase 01 1
Day 1-3

Gene expression modulation begins. Unlike fast-acting anxiolytics, Selank works through transcriptional changes requiring time for protein synthesis. Minimal acute effects expected as GABA receptor subunit genes begin modulation.

PMID:26924987
Phase 02 2
Week 1

Initial anxiolytic effects may emerge as GABA-A receptor subunit changes accumulate. Russian clinical protocols typically use 14-day courses. Enkephalin levels begin rising due to aminopeptidase inhibition.

PMID:18454096
Phase 03 3
Week 2

Clinical trials show significant HAM-A score reductions by end of 14-day treatment. BDNF upregulation contributing to cognitive improvements. Mood stabilization from serotonin receptor modulation.

PMID:18454096
Phase 04 4
Week 2-4

Sustained anxiolytic effects with continued administration. No tolerance development observed in clinical studies. Cognitive benefits become more apparent with improved memory consolidation.

PMID:25176261
Phase 05 5
Post-treatment

Effects may persist beyond treatment cessation due to gene expression changes. No withdrawal symptoms reported, distinguishing Selank from benzodiazepines. Duration of post-treatment benefit not well characterized.

Research-based observations

This timeline reflects observations from published clinical and preclinical studies. Individual responses may vary significantly. This is not a guarantee of effects or a dosing schedule. Consult qualified healthcare providers for personalized guidance.

Good Signs (7 indicators)
White to off-white lyophilized powder (cake or crystalline appearance)
Dissolves completely and quickly in bacteriostatic water
Clear, colorless solution after reconstitution
Comes with certificate of analysis (COA) showing >98% purity
Third-party HPLC and mass spectrometry verification available
Proper vacuum seal on vial before reconstitution
Nasal spray formulations should have clear delivery mechanism
Warning Signs (6 indicators)
Slightly off-white or cream-colored powder (may still be acceptable)
Takes longer than expected to fully dissolve
Powder appears collapsed or melted (possible moisture exposure)
COA from manufacturer only without third-party verification
Purity listed below 98% but above 95%
Nasal spray with inconsistent delivery
Bad Signs (7 indicators)
Yellow, brown, or otherwise discolored powder
Visible particles or cloudiness after reconstitution
Gel-like consistency or clumping that won't dissolve
No COA provided or COA appears fraudulent
Strong unusual odor
Vial seal appears compromised or previously opened
Nasal spray with visible contamination or broken mechanism
Positive quality indicator
Requires evaluation
Potential quality issue

For Research Evaluation Only

These quality indicators are general guidelines based on typical peptide characteristics. Professional laboratory testing (HPLC, mass spectrometry) provides definitive quality verification. This checklist is for initial visual evaluation only.

Synergistic
Compatible
Caution
Avoid

Semax

Synergistic
Synergistic

Both Russian regulatory peptides with complementary effects - Selank provides anxiolysis while Semax enhances cognition. Often used together in Russian clinical practice.

Both Russian bioregulator peptides with distinct targets - Selank for anxiety via GABA modulation, epithalon for longevity via telomerase. No known interactions.

Both have immunomodulatory properties - Selank via tuftsin backbone, Ta1 as primary function. May have complementary immune effects.

Different primary targets - Selank for CNS anxiolysis, BPC-157 for tissue repair. BPC-157's reported CNS effects may complement Selank's anxiolytic action.

LL-37

Compatible
Compatible

Selank retains immunomodulatory properties from tuftsin backbone; LL-37 provides direct antimicrobial effects. Different immune modulation pathways.

Research Note: Interaction data is based on published literature, mechanistic understanding, and theoretical considerations. Most peptide combinations lack direct clinical study. This information is for educational purposes only and does not constitute medical advice. Always consult qualified healthcare providers.

This dossier synthesizes available evidence from peer-reviewed literature, regulatory documents, and clinical trial registries. Evidence strength ratings follow a modified GRADE approach.

For complete methodology details, see our Methodology page.

Important Disclaimer

This dossier is for educational purposes only and does not constitute medical advice. Always consult a qualified healthcare provider before making health decisions.

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