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ID: STAMAKORT STATUS: ACTIVE

Stamakort

Research Only

Also known as: Gastric peptide, Stomach bioregulator, Gastric mucosa peptide

A peptide bioregulator developed by Russian scientist Vladimir Khavinson for gastric mucosa support. Claimed to modulate stomach lining gene expression and support digestive function. No Western clinical validation exists.

Other Very Low Evidence 6 Sources

Research Statistics

Total Sources
6
Human Studies
0
Preclinical
5
Evidence Rating Very Low Evidence
Research Depth 1/5
Global Coverage 1/5
Mechanism Plausibility 2/5
Overall Score
1.5 /5

Russian bioregulator (Khavinson lab); minimal published research even by Russian standards. Animal-only data from Russian Institute of Bioregulation. No human trials, no Western replication. Stomach tissue bioregulation mechanism is theoretical.

Last reviewed February 2026 How we rate →
!!
Evidence Level
very low
Not approved for human use by any regulatory agency
Limited human clinical trial data
Consult a healthcare provider before use
Not FDA Approved WADA Prohibited

Research Dossier

01 / 5

Overview

What is Stamakort and what does the research say?

Identity
Also Known As
Gastric peptide • Stomach bioregulator • Gastric mucosa peptide

Mechanism of Action

Stamakort is proposed to support gastric mucosa through peptide bioregulation mechanisms developed by Vladimir Khavinson.

Proposed Mechanisms

  1. Mucosal Gene Modulation - Claimed to regulate gene expression in gastric mucosa cells
  2. Protective Function - Proposed effects on stomach lining integrity
  3. Digestive Support - Claimed involvement in gastric secretory function

Important Limitations

  • No Western clinical validation or regulatory approval
  • Limited Russian preclinical data only
  • Amino acid sequence not fully characterized in public literature
  • NOT a treatment for ulcers or gastric conditions
  • No controlled human clinical trials

Evidence-Chained Benefits

Evidence-Chained Benefits

Research findings linked to mechanisms and clinical outcomes

Mechanism Gastric mucosa gene expression modulation
Emerging 2 direct studies
Benefit suggested to support gastric mucosa function
Evidence Level
Very Low
2 Animal
1 In Vitro
Mechanism Confidence
Established
Supported
Emerging
Evidence Level
High
Moderate
Low
Very Low

Peptide Interactions

Known and theoretical interactions when combining Stamakort with other peptides. Based on published research and mechanistic considerations.

Synergistic
Compatible
Caution
Avoid

Both target digestive system - Chonluten for GI tract broadly, Stamakort specifically for gastric mucosa.

Both Khavinson bioregulator peptides with distinct tissue targets.

Research Note: Interaction data is based on published literature, mechanistic understanding, and theoretical considerations. Most peptide combinations lack direct clinical study. This information is for educational purposes only and does not constitute medical advice. Always consult qualified healthcare providers.

References

6 Sources
0 Human
5 Preclinical

Key Studies Cited

Full reference list available on request. All citations link to PubMed for verification.

Methodology Note

This dossier synthesizes available evidence from peer-reviewed literature, regulatory documents, and clinical trial registries. Evidence strength ratings follow a modified GRADE approach.

For complete methodology details, see our Methodology page.

Important Disclaimer

This dossier is for educational purposes only and does not constitute medical advice. Always consult a qualified healthcare provider before making health decisions.

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