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A specimen of the Immune drawer Drawer F · Immune

Murepavadin

Investigational

A first-in-class cyclic antimicrobial peptide targeting the LptD outer membrane protein of Pseudomonas aeruginosa. The first OMPTA (outer membrane protein targeting antibiotic) to reach clinical development. IV formulation discontinued due to nephrotoxicity; inhaled formulation continues Phase 3 development for cystic fibrosis and bronchiectasis patients.

POL7080 · RG7929

Research evidence
Moderate

18 human studies

Preclinical
36%
Clinical
64%

Based on 28 cited sources

Evidence Score63/100
Emerging / moderate
Research Depth60/100
Mechanism95/100
Plausibility77/100
Global Coverage65/100
Community Experience5/100
EffectivenessNot Established

Demonstrated effect magnitude — not a recommendation or safety claim.

Research Depth 60 (1A 25: best published human evidence is small double-blind placebo-controlled Phase 1 PK/safety RCTs 29437621/30012756, n<50 per cohort; no published efficacy data — Phase 3 IV HABP/VABP program halted for nephrotoxicity 31554212, inhaled Phase 3 unreported; 1B 14 low-RoB on PK/safety only; 1C 6 total human N ~100-300; 1D 7 PK surrogate, right population but efficacy outcome never read out; 1E 8 substantial multi-model preclinical program). Mechanism 95 (2A 40: LptD target confirmed by binding + photo- crosslinking 29359918 and resistance/WGS lptD mutations 33367642; 2B 28 full LptD->LPS-transport-block->membrane-failure->lysis chain; 2C 17 MIC dose- response plus in-vivo AUC/MIC PK/PD index 30642931; 2D 10 confirmed in mouse models). Plausibility 77 (3A 26: mechanism->bactericidal surrogate established, surrogate->clinical cure inferred only; 3B 23 coherent with LPS-transport biology; 3C 14 anti-pseudomonal class analogy though first-in-class OMPTA; 3D 14 tightly scoped narrow-spectrum claim). Global Coverage 65 (4A 24 replicated by independent groups — JMI/US 29901750, Radboud-Erasmus/NL 30642931, Ramon y Cajal/ES 33367642, Emory/US 31554212 — beyond Zurich/Polyphor origin; 4B 20 many countries; 4C 16 ~20-30 study literature; 4D 5 investigational, trial-registered, never approved). Community Experience 5: investigational antibiotic with no documented real-world/community use. Effectiveness not-established: no quantified human efficacy estimate (Phase 3 IV halted pre-readout, inhaled Phase 3 pending) and no meaningful community effect signal.

Scored June 2026 How we rate →
~
Evidence Level
moderate
Not approved for human use by any regulatory agency
Limited human clinical trial data
Consult a healthcare provider before use
Not FDA Approved WADA Prohibited
Identity
SCALE · 1:1 N-TERMINUS C-TERMINUS 14 AA · 1,885 Da
Also Known As
POL7080 • RG7929
Class
Cyclic peptidomimetic
Length
14 amino acids
Mol. weight
1,885 Da
Sequence
Cyclic beta-hairpin (proprietary)
Molecular Structure
DPro
Pro
Arg
Arg
Tyr
Arg
Val
Trp
Cys
Arg
Arg
Tyr
Phe
Cys
Hydrophobic
Polar
Positive
Negative

Murepavadin is the first-in-class Outer Membrane Protein Targeting Antibiotic (OMPTA), specifically targeting the LptD protein in Pseudomonas aeruginosa. Its mechanism is supported by extensive structural and biochemical studies, including photo-crosslinking work that localized its binding site on LptD.

How It Works (Simplified)

Murepavadin acts as a precision weapon against P. aeruginosa by blocking a critical transport system:

LptD Binding

Binds to the N-terminal plug domain of LptD, the outer membrane protein that inserts lipopolysaccharide (LPS) into the bacterial membrane.

LPS Transport Block

Locks LptD in a non-functional conformation, preventing new LPS from being inserted into the outer membrane.

Membrane Disruption

Without new LPS, the bacterial outer membrane loses integrity and becomes asymmetric, leading to cell death.

Species Selectivity

Only P. aeruginosa LptD has the specific structure murepavadin targets, preserving other bacteria and having no effect on human cells.

Key Research: Andolina G et al. (Switzerland, 2018) mapped the LptD binding site using photo-crosslinking and mass-spectrometry-based proteomics, localizing the interaction to the Pseudomonas-specific periplasmic segment of LptD. PMID:29359918

Important Limitations

  • IV formulation development discontinued due to nephrotoxicity in Phase 3 trials
  • Only active against P. aeruginosa (not useful for polymicrobial infections)
  • Inhaled formulation still in Phase 3 trials (not yet approved)
  • Long-term safety data beyond 28 days of treatment not available
  • Resistance mechanisms exist (LptD mutations) though frequency is low
i. Lpt Transport Pathway · LPS Assembly
LPS synthesis (inner membrane)LptBFGC (ABC transporter)LptA (periplasm)Murepavadin ⟶ LptDE (blocked) ⟶ X No LPS insertionOuter membrane failure
ii. Bactericidal Mechanism · Cell Death
LptD inhibitionLPS accumulation in periplasmMembrane asymmetryCell lysis
Mechanism LptD inhibition blocking lipopolysaccharide transport to outer membrane
Established 8 direct studies
Benefit shown to kill Pseudomonas aeruginosa bacteria
Evidence Level
Moderate
3 Human
4 Animal
6 In Vitro
Mechanism Disruption of outer membrane integrity via LPS depletion
Established 5 direct studies
Benefit appears to treat drug-resistant P. aeruginosa infections
Evidence Level
Moderate
2 Human
3 Animal
4 In Vitro
Mechanism Species-specific targeting of P. aeruginosa LptD protein structure
Established 4 direct studies
Benefit may preserve beneficial microbiome during treatment
Evidence Level
Low
1 Human
2 Animal
3 In Vitro
Mechanism High local concentration delivery via inhaled formulation
Supported 3 direct studies
Benefit appears to reduce systemic toxicity while maintaining efficacy
Evidence Level
Low
1 Human
2 Animal
Mechanism Confidence
Established
Supported
Emerging
Evidence Level
High
Moderate
Low
Very Low
Phase 01 1
Hours 1-6

Based on Phase 1 data: peak plasma concentrations are achieved at the end of the IV infusion, with a geometric mean half-life of roughly 2.5-5.3 h and dose-proportional, linear pharmacokinetics.

PMID:29437621
Phase 02 2
Days 1-3

In vitro, murepavadin is bactericidal against most P. aeruginosa strains within 1-5 hours at higher concentrations, consistent with sustained exposure above the MIC.

PMID:33367642
Phase 03 3
Days 3-7

Exposure to murepavadin increases in renal impairment (AUC roughly 2- to 2.5-fold higher), so the elimination half-life lengthens over a multi-day course and dose adjustment is warranted with reduced renal function.

PMID:30012756
Phase 04 4
Days 7-14

With prolonged IV treatment, a nephrotoxicity signal led to discontinuation of the Phase 3 IV program; development has since focused on the inhaled formulation.

PMID:31554212

Research-based observations

This timeline reflects observations from published clinical and preclinical studies. Individual responses may vary significantly. This is not a guarantee of effects or a dosing schedule. Consult qualified healthcare providers for personalized guidance.

Good Signs (6 indicators)
Clear, colorless solution after reconstitution
Proper temperature-controlled shipping (cold chain)
Certificate of analysis from pharmaceutical manufacturer
GMP-compliant manufacturing documentation
Intact vial seal and sterility indicators
Within expiration date with appropriate lot tracking
Warning Signs (5 indicators)
Slight turbidity after reconstitution
Temperature excursion during shipping
Missing batch documentation
Near expiration date
Storage conditions unclear or non-compliant
Bad Signs (6 indicators)
Visible particles or discoloration
Compromised vial seal
No certificate of analysis or GMP documentation
Expired product
Unknown or unverified source
Evidence of freeze-thaw cycles
Positive quality indicator
Requires evaluation
Potential quality issue

For Research Evaluation Only

These quality indicators are general guidelines based on typical peptide characteristics. Professional laboratory testing (HPLC, mass spectrometry) provides definitive quality verification. This checklist is for initial visual evaluation only.

Synergistic
Compatible
Caution
Avoid

LL-37

Compatible
Compatible

Both are antimicrobial peptides with different mechanisms. LL-37 has broad immunomodulatory effects while murepavadin specifically targets P. aeruginosa LptD. No known contraindications.

Different antimicrobial mechanisms. Lactoferricin has membrane-disrupting activity while murepavadin inhibits LPS transport. May have complementary antibacterial effects.

Both target Gram-negative bacteria. Colistin's membrane disruption differs from murepavadin's LptD inhibition. Combined nephrotoxicity risk with IV formulations; monitor renal function.

Research Note: Interaction data is based on published literature, mechanistic understanding, and theoretical considerations. Most peptide combinations lack direct clinical study. This information is for educational purposes only and does not constitute medical advice. Always consult qualified healthcare providers.

This dossier synthesizes available evidence from peer-reviewed literature, regulatory documents, and clinical trial registries. Evidence strength ratings follow a modified GRADE approach.

For complete methodology details, see our Methodology page.

Important Disclaimer

This dossier is for educational purposes only and does not constitute medical advice. Always consult a qualified healthcare provider before making health decisions.

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